Diabetic ketoacidosis (DKA) is defined by the biochemical triad of ketosis, hyperglycemia and acidosis. The main treatment of DKA is rehydration, insulin
SGLT2‑i medicines are associated with an increased risk of DKA that does not always occur with significantly elevated blood glucose levels.4. That is, the DKA
In addition, common drugs that can trigger DKA include glucocorticoids, diuretics and atypical antipsychotics. DKA lab work-up DKA work-up should include CBC, electrolytes, extended electrolytes, creatinine, BUN, albumin, VBG, lactate, serum ketones, as well as consideration for:
Med Dose - Incr HR. High Dose - Incr BP. Epinephrine. 0.03-1 mcg/kg/min. 1-10 See Peds Protocol. (DKA vs non DKA). See Adult Protocol. (DKA vs non DKA).
DKA can be fatal without immediate intravenous insulin. Contact a healthcare provider right away if you develop DKA symptoms. 2. Heart and Blood
CONTENTS Rapid Reference Getting started Evaluating anion gap ketoacidosis Definition severity of DKA Evaluating the cause of DKA Core components of DKA resuscitation Fluid administration Electrolyte management Insulin infusion Long-acting, basal insulin Management of severe or refractory ketoacidosis NAGMA management Monitoring management of DKA recurrence Special situations DKA in a
The sulfonylureas are contraindicated in patients with a known hypersensitivity to the drug and in those with DKA, with or without coma. DKA should be treated
See Peds Protocol. (DKA vs non DKA). See Adult Protocol. (DKA vs non DKA) Nitroglycerin. 0.25-3 mcg/kg/min. (max 5 mcg/kg/min). mcg/min. Vasodilator.
Treatment of DKA involves volume expansion, insulin replacement, and prevention of hypokalemia. DKA can be prevented through the earlier recognition and
Gosh, may gayuma ka ata... :-D Super amused with the tinola! Hmmmn, dka nagluluto ano? Hehe.